Raimundo
Freire Betancor
Investigador Externo
University of Zurich
Zúrich, SuizaPublicaciones en colaboración con investigadores/as de University of Zurich (10)
2023
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Interferon restores replication fork stability and cell viability in BRCA-defective cells via ISG15
Nature Communications, Vol. 14, Núm. 1
2022
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Stress-triggered hematopoietic stem cell proliferation relies on PrimPol-mediated repriming
Molecular Cell, Vol. 82, Núm. 21, pp. 4176-4188.e8
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The CDK1-TOPBP1-PLK1 axis regulates the Bloom's syndrome helicase BLM to suppress crossover recombination in somatic cells
Science Advances, Vol. 8, Núm. 5
2019
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The p97–Ataxin 3 complex regulates homeostasis of the DNA damage response E3 ubiquitin ligase RNF8
EMBO Journal, Vol. 38, Núm. 21
2018
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Histone Ubiquitination by the DNA Damage Response Is Required for Efficient DNA Replication in Unperturbed S Phase
Molecular Cell, Vol. 71, Núm. 6, pp. 897-910.e8
2016
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Cullin3-KLHL15 ubiquitin ligase mediates CtIP protein turnover to fine-tune DNA-end resection
Nature Communications, Vol. 7
2005
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The human Rothmund-Thomson syndrome gene product, RECQL4, localizes to distinct nuclear foci that coincide with proteins involved in the maintenance of genome stabiliy
Journal of Cell Science, Vol. 118, Núm. 18, pp. 4261-4269
2004
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The human Rad9/Rad1/Hus1 damage sensor clamp interacts with DNA polymerase β and increases its DNA substrate utilisation efficiency: Implications for DNA repair
Nucleic Acids Research, Vol. 32, Núm. 11, pp. 3316-3324
2001
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Cleavage of the Bloom's syndrome gene product during apoptosis by caspase-3 results in an impaired interaction with topoisomerase IIIα
Nucleic Acids Research, Vol. 29, Núm. 15, pp. 3172-3180
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Direct association of Bloom's syndrome gene product with the human mismatch repair protein MLH1
Nucleic Acids Research, Vol. 29, Núm. 21, pp. 4378-4386