Publications (21)


  1. Reducing peripheral inflammation with infliximab reduces neuroinflammation and improves cognition in rats with hepatic encephalopathy

    Frontiers in Molecular Neuroscience, Vol. 9, Núm. NOV2016

  2. Neuroinflammation increases GABAergic tone and impairs cognitive and motor function in hyperammonemia by increasing GAT-3 membrane expression. Reversal by sulforaphane by promoting M2 polarization of microglia

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  3. Infliximab reduces peripheral inflammation, neuroinflammation, and extracellular GABA in the cerebellum and improves learning and motor coordination in rats with hepatic encephalopathy

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  4. In vivo administration of extracellular cGMP normalizes TNF-α and membrane expression of AMPA receptors in hippocampus and spatial reference memory but not IL-1β, NMDA receptors in membrane and working memory in hyperammonemic rats

    Brain, Behavior, and Immunity, Vol. 57, pp. 360-370

  5. Hyperammonemia induces glial activation, neuroinflammation and alters neurotransmitter receptors in hippocampus, impairing spatial learning: Reversal by sulforaphane

    Journal of Neuroinflammation, Vol. 13, Núm. 1

  6. Hyperammonemia alters glycinergic neurotransmission and modulation of the glutamate-nitric oxide-cGMP pathway by extracellular glycine in cerebellum in vivo

    Journal of Neurochemistry, Vol. 137, Núm. 4, pp. 539-548

  7. Extracellular cGMP Modulates Learning Biphasically by Modulating Glycine Receptors, CaMKII and Glutamate-Nitric Oxide-cGMP Pathway

    Scientific Reports, Vol. 6