Javier Rafael Castro Hernández-rekin lankidetzan egindako argitalpenak (11)

2021

  1. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

  2. Prolonged dopamine D3 receptor stimulation promotes dopamine transporter ubiquitination and degradation through a PKC-dependent mechanism

    Pharmacological Research, Vol. 165

2020

  1. DRD3 (dopamine receptor D3) but not DRD2 activates autophagy through MTORC1 inhibition preserving protein synthesis

    Autophagy, Vol. 16, Núm. 7, pp. 1279-1295

2016

  1. Long-term controlled GDNF over-expression reduces dopamine transporter activity without affecting tyrosine hydroxylase expression in the rat mesostriatal system

    Neurobiology of Disease, Vol. 88, pp. 44-54

2015

  1. Prolonged treatment with pramipexole promotes physical interaction of striatal dopamine D3 autoreceptors with dopamine transporters to reduce dopamine uptake

    Neurobiology of Disease, Vol. 74, pp. 325-335

2014

  1. Striatal vessels receive phosphorylated tyrosine hydroxylase-rich innervation from midbrain dopaminergic neurons

    Frontiers in Neuroanatomy, Vol. 8, Núm. AUG

2013

  1. Melatonin prevents dopaminergic cell loss induced by lentiviral vectors expressing A30P mutant alpha-synuclein

    Histology and Histopathology, Vol. 28, Núm. 8, pp. 999-1006

2010

  1. The dopamine transporter is differentially regulated after dopaminergic lesion

    Neurobiology of Disease, Vol. 40, Núm. 3, pp. 518-530

  2. The neuronal-specific SGK1.1 kinase regulates δ-epithelial Na + channel independently of PY motifs and couples it to phospholipase C signaling

    American Journal of Physiology - Cell Physiology, Vol. 299, Núm. 4

  3. Vulnerability of mesostriatal dopaminergic neurons in Parkinson's disease

    Frontiers in Neuroanatomy

2009

  1. Dopamine transporter glycosylation correlates with the vulnerability of midbrain dopaminergic cells in Parkinson's disease

    Neurobiology of Disease, Vol. 36, Núm. 3, pp. 494-508