Bioquímica, Microbiología, Biología Celular y Genética
Departamento
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Massachusetts General Hospital
Boston, Estados UnidosPublicaciones en colaboración con investigadores/as de Massachusetts General Hospital (21)
2021
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Integrative omics provide biological and clinical insights into acute respiratory distress syndrome
Intensive Care Medicine, Vol. 47, Núm. 7, pp. 761-771
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Plasma Insulin-like Growth Factor Binding Protein 7 Contributes Causally to ARDS 28-Day Mortality: Evidence From Multistage Mendelian Randomization
Chest, Vol. 159, Núm. 3, pp. 1007-1018
2020
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Plasma Levels of Proresolving and Prophlogistic Lipid Mediators: Association With Severity of Respiratory Failure and Mortality in Acute Respiratory Distress Syndrome
Critical Care Explorations, Vol. 2, Núm. 10, pp. E0241
2019
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Deconstructing ARDS Variability: Platelet Count, an ARDS Intermediate Phenotype and Novel Mediator of Genetic Effects in ARDS
Seminars in Respiratory and Critical Care Medicine, Vol. 40, Núm. 1, pp. 12-18
2018
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Erratum to: Genome-wide association and HLA fine-mapping studies identify risk loci and genetic pathways underlying allergic rhinitis (Nature Genetics, (2018), 50, 8, (1072-1080), 10.1038/s41588-018-0157-1)
Nature Genetics
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Genome-wide association and HLA fine-mapping studies identify risk loci and genetic pathways underlying allergic rhinitis
Nature Genetics
2017
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A missense genetic variant in LRRC16A/CARMIL1 improves acute respiratory distress syndrome survival by attenuating platelet count decline
American Journal of Respiratory and Critical Care Medicine, Vol. 195, Núm. 10, pp. 1353-1361
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Late-onset moderate to severe acute respiratory distress syndrome is associated with shorter survival and higher mortality: a two-stage association study
Intensive Care Medicine, Vol. 43, Núm. 3, pp. 399-407
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Whole blood microRNA markers are associated with acute respiratory distress syndrome
Intensive Care Medicine Experimental , Vol. 5, Núm. 1
2015
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Platelet count mediates the contribution of a genetic variant in LRRC16A to ARDS risk
Chest, Vol. 147, Núm. 3, pp. 607-617
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Predictors of circulating insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 in critical illness
Critical Care Medicine, Vol. 43, Núm. 12, pp. 2651-2659
2014
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Functional characterization of polymorphisms in the peptidase inhibitor 3 (Elafin) gene and validation of their contribution to risk of acute respiratory distress syndrome
American Journal of Respiratory Cell and Molecular Biology, Vol. 51, Núm. 2, pp. 262-272
2013
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IL 1RN coding variant is associated with lower risk of acute respiratory distress syndrome and increased plasma IL-1 receptor antagonist
American Journal of Respiratory and Critical Care Medicine, Vol. 187, Núm. 9, pp. 950-959
2012
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Distinct and replicable genetic risk factors for acute respiratory distress syndrome of pulmonary or extrapulmonary origin
Journal of Medical Genetics, Vol. 49, Núm. 11, pp. 671-680
2009
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Genetic polymorphisms of peptidase inhibitor 3 (elafin) are associated with acute respiratory distress syndrome
American Journal of Respiratory Cell and Molecular Biology, Vol. 41, Núm. 6, pp. 696-704
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Genetic variants in the angiopoietin-2 gene are associated with increased risk of ARDS
Intensive Care Medicine, Vol. 35, Núm. 6, pp. 1024-1030
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Heme Oxygenase-1 microsatellite polymorphism and haplotypes are associated with the development of acute respiratory distress syndrome
Intensive Care Medicine, Vol. 35, Núm. 8, pp. 1343-1351
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Serum bilirubin levels on ICU admission are associated with ARDS development and mortality in sepsis
Thorax, Vol. 64, Núm. 9, pp. 784-790
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Sex-specific association of epidermal growth factor gene polymorphisms with acute respiratory distress syndrome
European Respiratory Journal, Vol. 33, Núm. 3, pp. 543-550
2000
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Oligodendrocytes in brain and optic nerve express the β3 subunit isoform of Na,K-ATPase
GLIA, Vol. 31, Núm. 3, pp. 206-218