Publicaciones en las que colabora con Alfredo Santana Rodríguez (12)

2013

  1. LIF insensitivity and expression of proteins activated by DNA damage response in teratoma-isolated cells derived from mouse embryonic stem cells

    Cytologia, Vol. 78, Núm. 2, pp. 195-202

2011

  1. Phenotypic correction of a mouse model for primary hyperoxaluria with adeno-associated virus gene transfer

    Molecular Therapy, Vol. 19, Núm. 5, pp. 870-875

2008

  1. Apocrine carcinoma of the anogenital region. A case report including immunohistochemical and molecular study, discussion of differential diagnosis and a review of the literature [1]

    International Journal of Colorectal Disease

2006

  1. Alanine-glyoxylate aminotransferase-deficient mice, a model for primary hyperoxaluria that responds to adenoviral gene transfer

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 103, Núm. 48, pp. 18249-18254

  2. Attenuation of dextran sodium sulphate induced colitis in matrix metalloproteinase-9 deficient mice

    World Journal of Gastroenterology, Vol. 12, Núm. 40, pp. 6464-6472

  3. Increased activity and expression of gelatinases in ischemic colitis

    Digestive Diseases and Sciences, Vol. 51, Núm. 12, pp. 2393-2399

  4. Matrix metalloproteinase-9 modulates intestinal injury in rats with transmural colitis

    Journal of Leukocyte Biology, Vol. 79, Núm. 5, pp. 954-962

2005

  1. Feasibility of hepatocyte transplantation-based therapies for primary hyperoxalurias

    American Journal of Nephrology

  2. Induction of colonic transmural inflammation by Bacteroides fragilis: Implication of matrix metalloproteinases

    Inflammatory Bowel Diseases, Vol. 11, Núm. 2, pp. 99-105

2004

  1. Metaloproteinasas de matriz en enfermedades del tracto gastrointestinal

    Gastroenterologia y Hepatologia, Vol. 27, Núm. 8, pp. 491-497

2003

  1. Patología molecular de la hiperoxaluria: Primaria

    Nefrologia, Vol. 23, Núm. SUPPL.1, pp. 90-97

  2. Primary hyperoxaluria type 1 in the Canary Islands: A conformational disease due to I244T mutation in the P11L-containing alanine:glyoxylate aminotransferase

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 100, Núm. 12, pp. 7277-7282