Publicaciones en colaboración con investigadores/as de Massachusetts General Hospital (21)

2021

  1. Integrative omics provide biological and clinical insights into acute respiratory distress syndrome

    Intensive Care Medicine, Vol. 47, Núm. 7, pp. 761-771

  2. Plasma Insulin-like Growth Factor Binding Protein 7 Contributes Causally to ARDS 28-Day Mortality: Evidence From Multistage Mendelian Randomization

    Chest, Vol. 159, Núm. 3, pp. 1007-1018

2020

  1. Plasma Levels of Proresolving and Prophlogistic Lipid Mediators: Association With Severity of Respiratory Failure and Mortality in Acute Respiratory Distress Syndrome

    Critical Care Explorations, Vol. 2, Núm. 10, pp. E0241

2019

  1. Deconstructing ARDS Variability: Platelet Count, an ARDS Intermediate Phenotype and Novel Mediator of Genetic Effects in ARDS

    Seminars in Respiratory and Critical Care Medicine, Vol. 40, Núm. 1, pp. 12-18

2018

  1. Erratum to: Genome-wide association and HLA fine-mapping studies identify risk loci and genetic pathways underlying allergic rhinitis (Nature Genetics, (2018), 50, 8, (1072-1080), 10.1038/s41588-018-0157-1)

    Nature Genetics

  2. Genome-wide association and HLA fine-mapping studies identify risk loci and genetic pathways underlying allergic rhinitis

    Nature Genetics

2017

  1. A missense genetic variant in LRRC16A/CARMIL1 improves acute respiratory distress syndrome survival by attenuating platelet count decline

    American Journal of Respiratory and Critical Care Medicine, Vol. 195, Núm. 10, pp. 1353-1361

  2. Late-onset moderate to severe acute respiratory distress syndrome is associated with shorter survival and higher mortality: a two-stage association study

    Intensive Care Medicine, Vol. 43, Núm. 3, pp. 399-407

  3. Whole blood microRNA markers are associated with acute respiratory distress syndrome

    Intensive Care Medicine Experimental , Vol. 5, Núm. 1

2015

  1. Platelet count mediates the contribution of a genetic variant in LRRC16A to ARDS risk

    Chest, Vol. 147, Núm. 3, pp. 607-617

  2. Predictors of circulating insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 in critical illness

    Critical Care Medicine, Vol. 43, Núm. 12, pp. 2651-2659

2014

  1. Functional characterization of polymorphisms in the peptidase inhibitor 3 (Elafin) gene and validation of their contribution to risk of acute respiratory distress syndrome

    American Journal of Respiratory Cell and Molecular Biology, Vol. 51, Núm. 2, pp. 262-272

2013

  1. IL 1RN coding variant is associated with lower risk of acute respiratory distress syndrome and increased plasma IL-1 receptor antagonist

    American Journal of Respiratory and Critical Care Medicine, Vol. 187, Núm. 9, pp. 950-959

2012

  1. Distinct and replicable genetic risk factors for acute respiratory distress syndrome of pulmonary or extrapulmonary origin

    Journal of Medical Genetics, Vol. 49, Núm. 11, pp. 671-680

2009

  1. Genetic polymorphisms of peptidase inhibitor 3 (elafin) are associated with acute respiratory distress syndrome

    American Journal of Respiratory Cell and Molecular Biology, Vol. 41, Núm. 6, pp. 696-704

  2. Genetic variants in the angiopoietin-2 gene are associated with increased risk of ARDS

    Intensive Care Medicine, Vol. 35, Núm. 6, pp. 1024-1030

  3. Heme Oxygenase-1 microsatellite polymorphism and haplotypes are associated with the development of acute respiratory distress syndrome

    Intensive Care Medicine, Vol. 35, Núm. 8, pp. 1343-1351

  4. Serum bilirubin levels on ICU admission are associated with ARDS development and mortality in sepsis

    Thorax, Vol. 64, Núm. 9, pp. 784-790

  5. Sex-specific association of epidermal growth factor gene polymorphisms with acute respiratory distress syndrome

    European Respiratory Journal, Vol. 33, Núm. 3, pp. 543-550

2000

  1. Oligodendrocytes in brain and optic nerve express the β3 subunit isoform of Na,K-ATPase

    GLIA, Vol. 31, Núm. 3, pp. 206-218