Bioquímica, Microbiología, Biología Celular y Genética
Departamento
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Harvard University
Cambridge, Estados UnidosPublicaciones en colaboración con investigadores/as de Harvard University (24)
2024
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Charting a landmark-driven path forward for population genetics and ancient DNA research in Africa
American Journal of Human Genetics, Vol. 111, Núm. 7, pp. 1243-1251
2023
2021
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Editorial: Precision Medicine in Pulmonary Diseases—Same Principles, New Approach
Frontiers in Medicine
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Integrative omics provide biological and clinical insights into acute respiratory distress syndrome
Intensive Care Medicine, Vol. 47, Núm. 7, pp. 761-771
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Plasma Insulin-like Growth Factor Binding Protein 7 Contributes Causally to ARDS 28-Day Mortality: Evidence From Multistage Mendelian Randomization
Chest, Vol. 159, Núm. 3, pp. 1007-1018
2020
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Plasma Levels of Proresolving and Prophlogistic Lipid Mediators: Association With Severity of Respiratory Failure and Mortality in Acute Respiratory Distress Syndrome
Critical Care Explorations, Vol. 2, Núm. 10, pp. E0241
2019
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Deconstructing ARDS Variability: Platelet Count, an ARDS Intermediate Phenotype and Novel Mediator of Genetic Effects in ARDS
Seminars in Respiratory and Critical Care Medicine, Vol. 40, Núm. 1, pp. 12-18
2017
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A missense genetic variant in LRRC16A/CARMIL1 improves acute respiratory distress syndrome survival by attenuating platelet count decline
American Journal of Respiratory and Critical Care Medicine, Vol. 195, Núm. 10, pp. 1353-1361
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An epigenome-wide association study of total serum IgE in Hispanic children
Journal of Allergy and Clinical Immunology, Vol. 140, Núm. 2, pp. 571-577
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Whole blood microRNA markers are associated with acute respiratory distress syndrome
Intensive Care Medicine Experimental , Vol. 5, Núm. 1
2015
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B cell-activating factor an orchestrator of lymphoid follicles in severe chronic obstructive pulmonary disease
American Journal of Respiratory and Critical Care Medicine, Vol. 192, Núm. 6, pp. 695-705
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Platelet count mediates the contribution of a genetic variant in LRRC16A to ARDS risk
Chest, Vol. 147, Núm. 3, pp. 607-617
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Predictors of circulating insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 in critical illness
Critical Care Medicine, Vol. 43, Núm. 12, pp. 2651-2659
2014
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Functional characterization of polymorphisms in the peptidase inhibitor 3 (Elafin) gene and validation of their contribution to risk of acute respiratory distress syndrome
American Journal of Respiratory Cell and Molecular Biology, Vol. 51, Núm. 2, pp. 262-272
2013
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IL 1RN coding variant is associated with lower risk of acute respiratory distress syndrome and increased plasma IL-1 receptor antagonist
American Journal of Respiratory and Critical Care Medicine, Vol. 187, Núm. 9, pp. 950-959
2012
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Distinct and replicable genetic risk factors for acute respiratory distress syndrome of pulmonary or extrapulmonary origin
Journal of Medical Genetics, Vol. 49, Núm. 11, pp. 671-680
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Inflammation and Immune-Related Candidate Gene Associations with Acute Lung Injury Susceptibility and Severity: A Validation Study
PLoS ONE, Vol. 7, Núm. 12
2011
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Interleukin-1 receptor-associated kinase 3 gene associates with susceptibility to acute lung injury
American Journal of Respiratory Cell and Molecular Biology, Vol. 45, Núm. 4, pp. 740-745
2010
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Ure2 is involved in nitrogen catabolite repression and salt tolerance via Ca2+ homeostasis and calcineurin activation in the yeast Hansenula polymorpha
Journal of Biological Chemistry, Vol. 285, Núm. 48, pp. 37551-37560
2009
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Genetic polymorphisms of peptidase inhibitor 3 (elafin) are associated with acute respiratory distress syndrome
American Journal of Respiratory Cell and Molecular Biology, Vol. 41, Núm. 6, pp. 696-704